Przemyslaw P. McEwan, Ph.D.

Dr. Przemyslaw P. McEwan is an associate in the firm’s Business Law department and a member of its Intellectual Property group.

Peter assists clients with patent preparation, patent prosecution, patentability analysis, freedom-to-operate analysis, evaluating competitive landscapes, intellectual property due diligence, and general intellectual property counseling. Peter has experience in managing patent portfolios and developing domestic and foreign patent prosecution strategies.

Peter’s technical expertise involves neuroscience, machine learning, immunology, molecular biology, gene editing and gene therapies, and medical diagnostics.

Prior to joining Goodwin, Peter worked as a Patent Agent at a large national firm. Peter conducted his doctoral research at Rutgers University, where he investigated molecular and cellular mechanisms of traumatic brain injuries. While in graduate school, Peter was an NIH Biotechnology fellow, where he gained expertise in a biotechnology-related disciplines, developed fluency in the principles and methods of the biological, chemical, and physical sciences, and learned to translate basic scientific discoveries into technologies that advance patient care and benefit humanity. Following graduate school, Peter conducted his post-doctoral research at University of Pennsylvania, where he investigated the mechanisms of white matter conductivity changes following concussions utilizing in vitro models, as well as rodent and pig animal models. 

In graduate school, Peter received a number of fellowships, including National Science Foundation Innovation Corps fellowship, New Jersey Commission for Spinal Cord Research fellowship, New Jersey Commission on Brain Injury Research fellowship, National Institutes of Health under Ruth L. Kirschstein fellowship in Biotechnology, and Anne B. and James B. Letham fellowship.

• Co-Author, “Federal Circuit Continues to Dismantle Diagnostic Patents,” Troutman Pepper, August 4, 2022
• Song H, McEwan PP*, Ameen-Ali KE, Tomasevich A, Kennedy-Dietrich C, Palma A, Arroyo EJ, Dolle JP, Johnson VE, Stewart W, Smith DH. Concussion leads to widespread axonal sodium channel loss and disruption of the node of Ranvier. Acta Neuropathologica 2022, 144(5):967-985.
• Rodriguez AR, Anderson ED, O’Neill KM, McEwan PP*, Vigilante NF, Kwon M, Akum BF, Stawicki TM, Meaney DF, Firestein BL. Cytosolic PSD-95 interactor alters functional organization of neural circuits and AMPA receptor signaling independent of PSD-95 binding. Network Neuroscience 2021, 5(1):166-197.
• Peng J, Yi MH, Jeong H, McEwan PP*, Zheng J, Wu G, Ganatra S, Ren Y, Richardson JR, Oh SB, Wu LJ. The voltage-gated proton channel Hv1 promotes microglia-astrocyte communication and neuropathic pain after peripheral nerve injury. Mol Brain. 2021 Jun 28;14(1):99.
• Albayar AA, Roche A, Swiatkowski P*, Antar S, Ouda N, Emara E, Smith DH, Ozturk AK, Awad BI. Biomarkers in Spinal Cord Injury: Prognostic Insights and Future Potentials. Front Neurol. 2019 Jan 29;10:27.
• Swiatkowski P*, Sewell E, Sweet ES, Dickson S, Swanson RA, McEwan SA, Cuccolo N, McDonnell ME, Patel MV, Varghese N, Morrison B, Reitz A, Meaney DF, Firestein BL. Cypin: a novel target for traumatic brain injury research and therapies. Neurobiology of Disease 2018, 119:13-25.
• Patel MV, Swiatkowski P*, Kwon M, Rodriguez AR, Campagno K, Firestein BL. A Novel Short Isoform of Cytosolic PSD-95 Interactor (Cypin) Regulates Neuronal Development. Mol Neurobiol. 2018 Aug;55(8):6269-6281.
• Rodríguez AR, O'Neill KM, Swiatkowski P*, Patel MV, Firestein BL. Overexpression of cypin alters dendrite morphology, single neuron activity, and network properties via distinct mechanisms. J Neural Eng. 2018 Feb;15(1):016020.
• Swiatkowski P*, Nikolaeva I, Kumar G, Zucco A, Akum BF, Patel MV, D’Arcangelo G, Firestein BL. Role of Akt-independent mTORC1 and GSK3β signaling in sublethal NMDA-induced injury and the recovery of neuronal electrophysiology and survival. Scientific Reports 2017, 7:1539.
• Swiatkowski P*, Murugan M, Eyo UB, Wang Y, Rangaraju S, Oh SB, Wu LJ. Activation of microglial P2Y12 receptor is required for outward potassium currents in response to neuronal injury. Neuroscience 2016, 318:22-33.
• Hernandez K, Swiatkowski P*, Patel MV, Liang C, Dudzinski NR, Brzustowicz LM, Firestein BL. Overexpression of Isoforms of Nitric Oxide Synthase 1 Adaptor Protein, Encoded by a Risk Gene for Schizophrenia, Alters Actin Dynamics and Synaptic Function. Front Cell Neurosci. 2016 Feb 2;10:6.
• Eyo UB, Peng J, Swiatkowski P*, Mukherjee A, Bispo A, Wu LJ. Neuronal hyperactivity recruits microglial processes via neuronal NMDA receptors and microglial P2Y12 receptors after status epilepticus. Journal of Neuroscience 2014, 34:10528-40.
• Swiatkowski P*, Sesti F. Delayed pharyngeal repolarization promotes abnormal calcium buildup in aging muscle. Biochemical and Biophysical Research Communications 2013, 433: 354-7.

(* published under “Swiatkowski” or “McEwan”)